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(3) SQOR in the regulation of ferroptosis “SQOR utilizes both hydrogen sulfide and hydrogen selenide to reduce ubiquinone (CoQ 10 ) to ubiquinol (CoQ 10 H 2 )” Recent studies have expanded the list of cellular enzymes capable of reducing CoQ 10 and modulating ferroptosis, with SQOR emerging as a notable addition alongside FSP1 and DHODH

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The peptide functions as a precision senolytic agent by competing with endogenous FOXO4 for p53 binding, releasing the tumor suppressor protein to trigger apoptosis specifically in cells that have accumulated the senescence-associated secretory phenotype (SASP) markers

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